Endocannabinoids are produced by post-synaptic neurons but act on receptors (CB1 receptors) in presynaptic neurons. One type of neuron that is a target of endocannabinoids is glutamatergic neurons. a. CB1 agonists such as THC reduce calcium influx by blocking the activity of voltage-dependent calcium channels. What effect would this have on the release of glutamate? b. Glutamate causes the membrane of postsynaptic neurons to become hyperpolarized. How would THC be expected to affect the rate of nerve impulse transmission in postsynaptic neurons that make synapses with the glutamatergic neuron?