There is a special type of complementation called allelic complementation in which your answer to part c does not hold true; allelic complementation is caused by a specific type of pseudo-reversion in which the two mutant organisms each have a mutation in different parts of the same gene (for example, let’s consider geneX: mutant 1 has a mutant allele of geneX, say X-1, while mutant 2 has a different mutant allele of geneX, X-2). Could allelic complementation be an indicator that a protein acts as a homomultimer, why or why not?